"Hangxiety" is one of the primary things Cheers helps reduce. If you have any science-y background, read http://www.jneurosci.org/content/32/1/390. Or you can read more about it here, https://cheershealth.com/products/restore.
DHM, one of our main ingredients, binds to the same brain receptor alcohol binds to (i.e.: the GABAa). Long story short, a major cause (if not the biggest cause) of why a person feels so poorly is GABAa rebound. Because alcohol is a depressant when alcohol leaves the system, the opposite effect is excitation--but not a nice kind, rather anxiety, shakiness, and a more squirrel-like ability to focus on any one thing. By binding to the GABAa receptor, DHM reduces GABAa rebound, ultimately affecting the feeling of anxiety and feeling panicky.
This rebound is also what causes you to sleep so poorly a few hours after drinking (hence the getting up and not being able to really go back to sleep a few hours after alcohol consumption). That's partly why Cheers Restore is taken after the last alcoholic beverage and not the next morning. However, if any anxiety or panicky-feelings still exist in the morning, additional Restore capsules can be taken to keep satiating the effects of the rebound.
A more simple way to describe DHM is that it is similar in effect to "hair of the dog" or a bloody mary, but without introducing more alcohol into the system (which is a bad strategy as it only puts off the inevitable symptoms into the future).
Everyone's body/brain is a little different, some people feel mostly bad the next day due to a long duration of acetaldehyde exposure (it's the toxic byproduct of alcohol which must occur before the liver can clear what once was alcohol out of the blood). Others feel bad primarily because of GABAa rebound. Others are a mixture of the two along with other causes. The negative effects of alcohol are universal, but its differing degrees of effect on various people are all genetic.
Generally speaking, the 1,200 mg of DHM in Restore makes a huge difference in this aspect because of its specific effect on GABAa rebound.